We report here that the established anticancer ruthenium( III )
complex NAMI-A induces potent and selective cytotoxic e ff ects in
a few leukaemia cell lines. These results sound very surprising after
20 years of intense studies on NAMI-A, commonly considered as a
“ non-cytotoxic ” antimetastatic agent. In addition, evidence is
given for selective inhibition of KCa 3.1 channels. The implications
of these fi ndings are discussed.
