The pattern of cognitive deficits in Parkinson's disease is variable, but often includes executive impairment similar to that seen in patients with frontal lesions, as well as episodic memory impairment, visuospatial dysfunction and impaired verbal fluency. That could derive from the not irrelevant "new" role of the basal ganglia in determining cognitive process refinement. We discuss a huge number of pathological observations, starting from neuro-anatomical and biochemical networks, from basal ganglia to thalamus and cortical cortex.
